CONCLUSION: Additive mechanisms from both the ICL and Adderall^(®) were present in our patient. The ICL caused crowding of the ACA through a pseudophacomorphic mechanism, and the Adderall^(®) caused increased iridotrabecular contact secondary to pharmacologic mydriasis. This resulted in episodic angle closure with subsequent spikes in the intraocular pressure. There are no current reports or studies in the current literature describing the combined mechanisms of ICL implantation and Adderall^(®)…
